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Research Which Confirms the Toxic Gas Explanation for Crib
Death
Publication
of the toxic gas theory:
- The toxic gas theory for cot death
(SIDS, crib death) was published by British scientist Barry Richardson in
1994: Sudden Infant Death Syndrome: a possible primary cause, Journal of
the Forensic Science Society 1994;34(3):199-204
Publication
of proof of the gas generation involved:
The fungal generation of extremely toxic nerve gases from compounds of
phosphorus, arsenic and antimony has been demonstrated many times over the last
century. Recent confirmations are:
- Toxic gas generation from plastic
mattresses and sudden infant death syndrome, Lancet 1995;346:1516-20
- Confirmation of the Biomethylation
of Antimony Compounds, Applied Organometallic Chemistry 1997; Vol.
11, 471-483
Publication of proof of the presence of fungal
growth in babies' mattresses:
- Sudden Infant Death Syndrome: a
possible primary cause, Journal of the Forensic Science Society
1994;34(3):199-204
- Final Report of the Expert Group to
Investigate Cot Death Theories: Toxic Gas Hypothesis (Limerick Report), May
1998
Publication
of proof of the presence of the elements phosphorus, arsenic and antimony in
babies' bedding:
-
Analyses of bedding reported by Jim
Sprott in The Cot Death Cover-up? (Penguin Books, New Zealand 1996,
Britain 1997).
Publication
of proof of the generation of highly toxic gases from compounds of phosphorus,
arsenic and antimony in babies' mattresses and other bedding:
- Sudden Infant Death Syndrome: a
possible primary cause, Journal of the Forensic Science Society
1994;34(3):199-204
- The evolution of phosphine from cot
mattress materials, M G Fitzpatrick, Department of Chemistry, University of
Auckland (Final Report, December 1997)
- Arsenic methylation by
micro-organisms isolated from sheepskin bedding materials, Human &
Experimental Toxicology (2003) 22:325-334
Publication of proof that the
re-use of mattresses increases the risk of cot death (a corollary of the toxic
gas theory):
- Case-control study of sudden infant
death syndrome in Scotland, 1992-5, British Medical Journal
1997;314:1516-20
- Used infant mattresses and sudden
infant death syndrome in Scotland: case-control study, British Medical
Journal 2002;325:1007-1009
Proof that
the risk of cot death rises from first to second babies in families; and from
second to third babies; and from third to fourth and later babies; and that
babies of solo parents are at higher risk again (a corollary of the fact that
re-use of mattresses increases the risk of cot death):
- Analysis of official British cot
death statistics (Source: Office of National Statistics, United Kingdom)
The finding of the rising risk of cot death from one sibling to the next
refutes every proposition that cot death has a medical cause. The more times
an unwrapped mattress is used from one baby to the next, the greater is the
risk of cot death.
This accounts for the higher cot death rate among poorer families, who are
more likely to sleep their babies on previously used mattresses.
If a mattress contains any of the elements phosphorus, arsenic or antimony,
and if certain household fungi have become established in the mattress
during prior use, any generation of toxic gas commences sooner and in
greater volume.
Publication
of proof that later babies in a family are more at risk of cot death than first
babies (a corollary of the fact that re-use of mattresses increases the risk of
cot death):
- Risk factors of sudden infant death
in Chinese babies, American Journal of Epidemiology 1997;144:1070-73
Publication
of proof that face-up sleeping reduces the risk of cot death:
- All studies which show that face-up
sleeping reduces the risk of cot death (of which there are many) support the
toxic gas theory. Face-up sleeping reduces the risk because the gases
concerned are more dense than air; they diffuse away from a baby's mattress
towards the floor, so a baby sleeping face-up is less likely to ingest them.
Publication
of proof that cot death babies show physiological effects of gaseous poisoning:
- Decreased Kainate Receptor Binding
in the Arcuate Nucleus of the Sudden Infant Death Syndrome, Journal of
Neuropathology and Experimental Neurology 1997;56:1253-61
- Synaptic Neurochemistry of Human
Striatum During Development: Changes in Sudden Infant Death Syndrome,
Journal of Neurochemistry 1993;60(6):2098-2105
Publication
of proof that the cot death risk varies with mattress type:
- New Zealand Cot Death Study
(1987-1990)
Publication
of proof that the cot death risk in Britain is less on PVC-covered mattresses:
- CESDI study (Britain), reported in
Lancet 1995;345:720
Any part of a baby's mattress which contains the chemicals phosphorus,
arsenic and/or antimony is capable of the gas generation which causes cot
death.
If, therefore, a mattress is covered with a gas-impermeable diaphragm which
does not contain those chemicals, the risk of cot death is eliminated.
Accordingly, following removal of those chemicals from British
plastic-covered mattresses from 1989 onwards, the risk of cot death in
Britain is less on PVC-covered mattresses (which are very frequently used in
Britain).
Thus the finding by the CESDI study that babies are less at risk on
PVC-covered mattresses supports the toxic gas theory for cot death.
Book
regarding the toxic gas theory for cot death
- Jim Sprott, The Cot Death
Cover-up? (Penguin Books, New Zealand 1996, Britain 1997).
Publication
of statistical results of the New Zealand mattress-wrapping campaign:
- Giftige Gase im Kinderbett (Toxic
Gases in Infants' Beds), Zeitschrift fuer Umweltmedizin (Journal of
Environmental Medicine) 2002; 44:18-20. See
BabeSafe Success.
- Cot Death - Cause and Prevention:
Experiences in New Zealand 1995-2004, Journal of Nutritional &
Environmental Medicine 2004; 14(3):221-232
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